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Blocking a specific immune molecule in the lungs of mice can alleviate asthma-related obesity symptoms in mice, which was the conclusion of a previous study in Nature-Medical.
Obesity is a risk factor for asthma, and an elevated body mass index is closely related to the higher risk of asthma. Although weight loss can alleviate the symptoms of asthma, the current asthma medications have little effect on obese people, and the factors of asthma in these people are still unknown.
Dale Umetsu et al. Reported that feeding on a high-fat diet causes mice to produce airway hyperresponsiveness, one of the basic symptoms of asthma. A high-fat diet will induce an immune cell in mice-macrophages to produce the immune system molecule interleukin-1b (IL-1b), which in turn promotes another molecule interleukin-17 (IL-17) Secretion eventually makes airway disease worse. A clinical drug that blocks the binding of IL-1b to its receptor can prevent the release of IL-17 and alleviate airway disease in obese mice. Since IL-17-producing cell types have been found in the airways of asthma patients, the conclusion of this study means that the treatment of this type of obese asthma may use IL-1b pathway as a breakthrough point.
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